Celiac Disease & Gluten-free Diet Information at Celiac.com

Celiac Disease & Gluten-free Diet Information at Celiac.com

Celiac Disease & Gluten-free Diet Information at Celiac.com – ArticlesCeliac Disease & Gluten-free Diet Information at Celiac.com – ArticlesBIORAY Kids for Organ SupportType 1 Diabetes – A Case Study Supporting Integration of Existing HypothesesMany College Students Struggle with Gluten-free Diet on Campus

http://ftr.fivefilters.org/makefulltextfeed.php?url=http%3A%2F%2Fwww.celiac.com%2Farticlerss.php&max=5 Celiac disease and gluten-free diet information at Celiac.com. Celiac disease, also known as gluten intolerance, is a genetic disorder that affects at least 1 in 133 Americans. Symptoms of celiac disease can range from the classic features, such as diarrhea, weight loss, and malnutrition, to latent symptoms such as isolated nutrient deficiencies but no gastrointestinal symptoms. 20 http://ftr.fivefilters.org/makefulltextfeed.php?url=http%3A%2F%2Fwww.celiac.com%2Farticlerss.php&max=5 http://www.celiac.com/apple-icon-180×180.png https://www.celiac.com/articles/24068/1/BIORAY-Kids-for-Organ-Support/Page1.html https://www.celiac.com/articles/24068/1/BIORAY-Kids-for-Organ-Support/Page1.html <p>”Nearly 30% of Americans avoid gluten to improve their overall health”, according to one poll(1). Gluten, a protein found in wheat, rye, spelt and barley, can cause health and mental symptoms in kids with genetic issues or immune sensitivities. There are ways to help such as choosing a gluten free lifestyle. Did you know there is also a new line of supplements to help too? BIORAY Kids™ support healthy organ function and remove toxins.</p> <p><strong>Undigested Food and <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;Malabsorption&lt;/b&gt;&lt;br&gt;&lt;br&gt;Abnormal absorption of nutrients from the digestive tract.’); return false”>Malabsorption</a></strong><br/>A major issue with children that have gluten sensitivities is they are prone to <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;malabsorption&lt;/b&gt;&lt;br&gt;&lt;br&gt;Abnormal absorption of nutrients from the digestive tract.’); return false”>malabsorption</a> of nutrients and ‘leaky gut’(2). This is when undigested food particles, <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;bacteria&lt;/b&gt;&lt;br&gt;&lt;br&gt;A large group of single-cell microorganisms. ‘); return false”>bacteria</a>, toxins and other waste leak into their bloodstream, and congest their organs. It’s no wonder kids get so irritable, have tummy aches or skin issues and experience brain fog; they are nutrient deficient and full of toxins!</p> <p><strong>BIORAY Kids Formulas Support Healthy, Happy Kids</strong><br/>Each BIORAY Kids™ product contains Natural Detox Factors (NDF®). The main ingredient is micronized chlorella that sticks to toxins and carries them safely out of the body. Research shows chlorella, significantly reduces leaky gut, and avoids gut <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;flora&lt;/b&gt;&lt;br&gt;&lt;br&gt;The microorganisms (as bacteria or fungi) live in or on the body, for example the flora of the intestines which aid in digestion.’); return false”>flora</a> and toxins from leaking into the bloodstream(3). NDF® is a rich source of nutrients too!</p> <p><a href=”https://www.celiac.com/adserv/www/delivery/ck.php?oaparams=2__bannerid=1775__zoneid=316__cb=f2a08044df__oadest=http%3A%2F%2Fbioraykids.com” rel=”nofollow” target=”_blank”><img title=”BIORAY Kids” src=”https://www.celiac.com/adserv/www/delivery/avw.php?zoneid=316&amp;cb=INSERT_RANDOM_NUMBER_HERE&amp;n=ab9e329f” alt=”BIORAY Kids”/></a>In addition to NDF®, each BIORAY Kids™ formula contains a unique blend of herbs and <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;probiotic&lt;/b&gt;&lt;br&gt;&lt;br&gt;General term for a substance that promotes the growth of microorganisms.’); return false”>probiotic</a> lysates to address specific issues. These formulas can be used individually or all together.</p> <ul><li>NDF CALM™ – For children who are grumpy, can’t sleep, easily frustrated or anxious.</li> <li>NDF SHINE™ – For kids who have a nervous stomach, <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;bowel&lt;/b&gt;&lt;br&gt;&lt;br&gt;The portion of the digestive tract between the stomach and the anus.’); return false”>bowel</a> issues, who are non-social or have delayed speech.</li> <li>NDF FOCUS™ – For kids who have problems focusing, brain fog, lack of energy, or seasonal histamine responses.</li> <li>NDF HAPPY™ – For children who have unreasonable anger, itchy rectums, crave sugar or grind their teeth at night.</li> </ul><p><strong>For more info visit their site: <a href=”https://www.celiac.com/adserv/www/delivery/ck.php?oaparams=2__bannerid=1775__zoneid=316__cb=f2a08044df__oadest=http%3A%2F%2Fbioraykids.com” rel=”nofollow” target=”_blank”>bioraykids.com</a></strong></p> <p>Sources:</p> <ol><li><a href=”https://www.npd.com/wps/portal/npd/us/news/press-releases/percentage-of-us-adults-trying-to-cut-down-or-avoid-gluten-in-their-diets-reaches-new-high-in-2013-reports-npd/” target=”_blank”>npd.com</a></li> <li><a href=”http://www.ncbi.nlm.nih.gov/pubmed/21822909″ target=”_blank”>nih.gov</a></li> <li><a href=”http://www.researchgate.net/publication/26658732_Administration_of_Chlorella_sp._microalgae_reduces_endotoxemia_intestinal_oxidative_stress_and_bacterial_translocation_in_experimental_biliary_obstruction” target=”_blank”>researchgate.net</a></li> </ol><p>Celiac.com welcomes your comments below (registration is NOT required).</p> <br/><div class=”ArticleExtra”> <h2><strong>Related Articles</strong></h2> </div> <br/><p><strong><a href=”https://blockads.fivefilters.org”></a></strong> <a href=”https://blockads.fivefilters.org/acceptable.html”>(Why?)</a></p> Tue, 24 Jan 2017 21:30:00 +0000 no@spam.com (Advertising Banner-Ads) BIORAY Kids for Organ Support – Celiac.com Article &quot;Nearly 30% of Americans avoid gluten to improve their overall health&quot;, according to one poll1. Gluten, a protein found in wheat, rye, spelt and barley, can cause health and mental symptoms in kids with genetic issues or immune sensitivities. There are ways to help such as choosing a gluten free lifestyle. Did you know there is also a new line of supplements to help too? BIORAY Kids™ support healthy organ function and remove toxins. https://www.celiac.com/articles/24068/1/BIORAY-Kids-for-Organ-Support/Page1.html https://www.celiac.com/content_images/Bioray-Kids_stores_page_plan_c.jpg de text/html https://www.celiac.com/articles/24068/1/BIORAY-Kids-for-Organ-Support/Page1.html https://www.celiac.com/articles/24660/1/Type-1-Diabetes–A-Case-Study-Supporting-Integration-of-Existing-Hypotheses/Page1.html https://www.celiac.com/articles/24660/1/Type-1-Diabetes–A-Case-Study-Supporting-Integration-of-Existing-Hypotheses/Page1.html <div itemprop=”image”><span class=”FeatureImageSpanArticle”><img src=”https://www.celiac.com/content_images/diabetes_CC–Jill_Brown_thumb.jpg” property=”og:image” class=”Picture” width=”600″ height=”600″/></span><br/><span class=”FeatureImageSpanArticle”>Image: CC–Jill Brown</span></div> <p>Celiac.com 01/24/2017 – <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;Diabetes&lt;/b&gt;&lt;br&gt;&lt;br&gt;A disease associated with the absence or reduced levels of insulin, a hormone essential for the transport of glucose to cells.’); return false”>Diabetes</a> is a condition in which blood glucose rises high enough to cause: damage to blood vessel walls, <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;neurological&lt;/b&gt;&lt;br&gt;&lt;br&gt;Of or relating to a branch of medicine concerned especially with the structure, functions, and diseases of the nervous system.’); return false”>neurological</a> injury, vision loss, and a host of other <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;maladies&lt;/b&gt;&lt;br&gt;&lt;br&gt;Diseases or sicknesses.’); return false”>maladies</a>. Most currently recognized cases of <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;diabetes&lt;/b&gt;&lt;br&gt;&lt;br&gt;A disease associated with the absence or reduced levels of insulin, a hormone essential for the transport of glucose to cells.’); return false”>diabetes</a> fall into one of two categories which are identified as type 1 and type 2 diabetes. While these two types of diabetes share many symptoms, the underlying causes are, in most cases, quite distinct, although there is also some overlap which will be explored shortly. There are also cases of gestational diabetes and some researchers are now suggesting that type 3 diabetes may be yet another entity that causes accelerating cell death in the brain, resulting dementia (1) but these latter two types of this condition are not included in the current discussion.</p> <p>All but one of these forms of diabetes involves cellular resistance to the action of insulin, although there is some gray area between type 1 and type 2 diabetes. Type 1 diabetes is the result of an <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;autoimmune&lt;/b&gt;&lt;br&gt;&lt;br&gt;A condition where the body’s immune system is unable to distinguish between foreign particles and the body’s own cells and as a result attacks normal body tissue.’); return false”>autoimmune</a> attack on a specific group of pancreatic cells called islets of Langerhans. These are the cells that produce insulin, a hormone that moves glucose out of the bloodstream and into various cells. About 14% of type 2 diabetics are also thought to experience a late-onset, slowly developing damage to pancreatic islet cells, which results in reduced insulin production in combination with their insulin resistance(2). This may be caused by autoimmunity, similar to type 1 diabetes, or it may be damage induced by other factors. Nonetheless, while type 2 diabetes can often be controlled either during weight loss or by reduced carbohydrate consumption alone, type 1 diabetes is not typically viewed as a condition that can be remedied by a change in eating habits. Yet there are some hints in the literature suggesting that dietary interventions may be therapeutically useful, especially if begun early enough in the disease process.</p> <p>Researchers Amanda MacFarlane and Fraser Scott report that there are several environmental factors, including specific foods, as well as viral, bacterial, and chemical agents that have been hypothesized to incite an autoimmune attack on the islet cells (2). They also report that about half of the animals that develop type 1 diabetes are mounting an immune response to wheat, which may also be involved in the attack on the insulin producing cells of the pancreas by either or both of two pathways they outline (2, 3). These hypothesized biological processes are identified as <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;molecular&lt;/b&gt;&lt;br&gt;&lt;br&gt;Relating to or produced by the simplest structural unit of an element or compound that retains all the properties of the substance and is composed of one or more atoms.’); return false”>molecular</a> <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;mimicry&lt;/b&gt;&lt;br&gt;&lt;br&gt;The act of mimicking or imitative behavior.’); return false”>mimicry</a> or bystander activation and cell death. While these authors favor bystander activation, either or both of these pathways may lead to an autoimmune attack on pancreatic islet cells. Regardless of the specific biological route, type 1 diabetes can be induced in a significant portion of genetically susceptible rats and mice, simply by feeding them a diet dominated by wheat gluten. Further, the severity of their disease varies directly with the proportion of wheat gluten in the diet (2). These investigators go on to say that “These similarities between coeliac disease in humans and diabetes in BB rats, NOD mice and type 1 diabetic patients are consistent with the idea that wheat is involved in diabetes <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;pathogenesis&lt;/b&gt;&lt;br&gt;&lt;br&gt;The cause or origin and subsequent development of a disease.’); return false”>pathogenesis</a>, possibly by inducing a subclinical, gut inflammation in many individuals that develop this form of diabetes” (2).</p> <p>They go on to report that: “Our data suggest that dietary modulation has effects at two (or more) levels:<br/>At the target cells before classic <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;insulitis&lt;/b&gt;&lt;br&gt;&lt;br&gt;Insulitis is an inflammation of the islets of Langerhans of the pancreas and this condition causes the Pancreatic β-cells to become infiltrated by mononuclear cells, which can lead to inflammation.’); return false”>insulitis</a>, changing the growth pattern of insulin-producing cells, enhancing islet mass and changing metabolism and insulin reserves . Dampening an ongoing inflammatory condition in the gut.” (2)</p> <p>Scott’s work (4, 5) along with investigations conducted by several groups of his colleagues (6-10) indicate that significant numbers of diabetes patients show immune reactions to the prolamins which are storage proteins in wheat, rye, and barley. Further, investigators have long understood that there is significant overlap between celiac disease and type 1 diabetes, with estimates ranging between 5% and 12% in each disease group (2, 11). MacFarlane and Scott point out that 33% to 40% of patients with type 1 diabetes show <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;transglutaminase&lt;/b&gt;&lt;br&gt;&lt;br&gt;A clotting factor that is a variant of factor XIII and that promotes the formation of cross-links between strands of fibrin.’); return false”>transglutaminase</a> autoantibodies which are similar to those found in celiac patients but usually at lower levels (2).</p> <p>Low concordance rates in monozygotic (identical) twins also suggest that environmental factors play a large role in causing type 1 diabetes (2). Again, the most compelling evidence indicates that dietary consumption of wheat gluten and similar prolamins is an important factor in the autoimmune attack that destroys the pancreatic capacity to produce insulin, in genetically susceptible individuals.</p> <p>Indirect support for this perspective is offered by animal research published in July of 2011. It shows that gamma-Aminobutryic acid (GABA) supplements not only inhibit the autoimmune attack on islet cells, GABA also incites regeneration of insulin producing cells (12). GABA is a non-toxic substance that is produced by the beta cells of the pancreas (13). It plays an inhibitory role throughout the nervous system which may be significant when taken in conjunction with Rodney Ford’s identification of gluten as the agent which, directly and indirectly, induces neurological damage in those with celiac disease and those with non-celiac gluten sensitivity. One pathway Ford identifies is gluten-induced neuronal excitation leading to cellular self-destruction. In light of Ford’s hypothesis, the inhibitory role of GABA on neuronal tissues, both at and near synapses, offers an inviting new window for envisioning the process that incites, and therefore may reverse, type 1 diabetes.</p> <p>Clearly there is considerable cause to suspect gluten grain consumption as an important factor in the onset and perpetuation of many cases of type 1 diabetes. While genetically coded <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;HLA&lt;/b&gt;&lt;br&gt;&lt;br&gt;Human Leukocyte Antigen – elicit the strongest immunologic response in the body, and chromosome 6 is the genetic region that codes for these antigens. The genes that encode the class II molecules DQ2 and DQ8, the key genetic risk factors in celiac disease. Because the HLA complex is inherited intact as two haplotypes (one from each parent), siblings have 1 in 4 chance of being HLA-identical.’); return false”>HLA</a> markers predispose to the disease, and a number of other environmental factors may play a role in its pathogenesis, prolamins from wheat and its close relatives are clearly a frequent and important contributor to this life-long condition in which exogenous insulin (injection with hypodermic needles) is necessary for maintaining optimal health (12) while living with this <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;malady&lt;/b&gt;&lt;br&gt;&lt;br&gt;A disease or sickness.’); return false”>malady</a>. However, given the insights offered by the above, the following case history may offer insights that might otherwise incite only scepticism. MacFarlane and Scott suggest the following: “One approach to achieving this [prevention] is to understand and modify the environmental factors that induce disease or equip those at risk with better means of avoiding or handling these agents”(2).</p> <p><strong>Case Study:</strong><br/>On January 18, 2008, three year old K and her anxious mother were taken to a hospital emergency department in Gilbert, AZ, where the attending physician concluded that the child had experienced a febrile seizure of about 5 minutes’ duration. At examination, she had a 102.5 degree temperature. In addition to fevers, K complained of abdominal pain and showed abdominal bloating. During this examination of K, she vomited. Laboratory tests showed elevated glucose (133 mg/dl) and an elevated white blood cell count (19,000). Tylenol was used to bring K’s temperature down and she was discharged with instructions for the parents to administer more Tylenol as needed, and to follow up with her regular health care provider within two days.</p> <p>By February 29, K experienced more fevers, ranging between 101 and 104, intermittently over 24 hours. Every four hours, when the effects of the previous dose of Tylenol wore off, the fever would, again, spike to 103-104. K was taken to see her regular physician the following day and urinalysis revealed ketone bodies. K and her parents were then sent to the emergency department of Banner Children’s Hospital.</p> <p>At the hospital, testing showed elevated urinary ketone bodies in the Large category, and blood showed elevated glucose at 193 mg/dL. Type 1 diabetes was diagnosed and K was admitted to hospital where she stayed for four days. Her condition was stabilized with ½ unit of Novalog and 4 units of Lantus. Meanwhile parents were educated about type 1 diabetes, insulin measurement and injection. They were taught to inject 1 unit of insulin for every 20 grams of carbohydrates consumed (20:1 ratio). K’s parents repeatedly wondered, in the presence of the diagnosing endocrinologist, just how much insulin K was producing and how many carbohydrates a thirty pound child needed to be healthy? *</p> <p>K’s father has a history of joint pain when consuming gluten grains. K was still experiencing abdominal bloating and because of the overlap between type 1 diabetes and celiac disease (2) <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;serum&lt;/b&gt;&lt;br&gt;&lt;br&gt;The liquid portion that remains when blood is allowed to clot spontaneously and is then centrifuged to remove the blood cells and clotting elements. It has approximately the same volume (55%) as plasma and differs from it only by the absence of fibrinogen. ‘); return false”>serum</a> IgA <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;antibody&lt;/b&gt;&lt;br&gt;&lt;br&gt;A high molecular weight protein which is produced by specialized B cells in the lymph nodes after stimulation by an antigen which acts specifically against the antigen in an immune response. They typically consist of four sub-units that include two heavy and two light chains. Also known as immunoglobulin.’); return false”>antibody</a> tests were undertaken and both transglutaminase and <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;gliadin&lt;/b&gt;&lt;br&gt;&lt;br&gt;A prolamin or simple protein that can be obtained by alcoholic extraction of gluten from wheat and rye, which has been identified as the culprit which causes intestinal damage in those with celiac disease.’); return false”>gliadin</a> antibody tests were negative. However, the parents observed that variations in the types of food K ate seemed to have a greater impact on blood glucose than a specific food’s putative sugar content.</p> <p>In keeping with their observations that different foods, despite their equal sugar content, produced different blood glucose results, the father’s history of joint pain when eating gluten, K’s abdominal bloating, and the widely documented connection between gluten grains and type 1 diabetes, these foods and several others were eliminated from her diet.</p> <p>K’s parents were quickly able to adjust the insulin therapy to a 40:1 ratio while K typically maintained a blood glucose range of between 80 and 95 mg/dl, which is well within the reference range for a healthy, non-diabetic person. In fact, this is a far narrower range than is prescribed by the American Diabetes Association which is 70-120 mg/dl for diabetic patients. K’s family continued to target and achieve the 80-95 mg/dl range.</p> <p>After a few months of lower than normal blood sugars, still on insulin therapy, with the carbohydrate ratio now 40/1, the parents sought permission from the endocrinologist to take K off insulin completely, on the condition that her blood sugar continued within the normal range of 85-95 mg/dl. This was monitored on a daily basis. The first 24 hours were a success and another day was granted.<br/>After six months of following a strict and intense food therapy diet for K, the family started reintroducing foods. Some foods were reintroduced without a rise in blood sugar. She was also able to eat a larger amount of carbohydrate each meal with the same blood sugar control. Clearly, the pancreas was producing increasing quantities of insulin.</p> <p>On August 21, 2008, six months into this intensive and individualized food therapy, the patient’s blood test results indicated a regeneration of the pancreas and a complete reversal of her type 1 diabetes. Her A1C was 4.8, well within the normal range for a non-diabetic person.</p> <p>Today, more than three years later, the patient is still insulin free and is using food therapy alone to maintain healthy and normal glucose control. Signs of pancreatic inflammation were also absent. Each of these findings echo MacFarlane and Scott on the issue of dietary intervention in animal studies.</p> <p>The intensive food therapy has now been replaced with a maintenance program. The variety of foods the patient can eat is vast. However, grain and casein continue to be avoided. It appears that, in this case, these foods may have contributed to K’s Type 1 diabetes. It may also be that the underlying cause of the fever K experienced early in this process was a factor in the onset of her type 1diabetes, and the transient nature of this fever, and its cause, may be at the root of her recovery from this ailment. Nonetheless, given the many converging research findings indicting grains and dairy proteins, along with K’s suggestive signs and symptoms, and her father’s reactions to gluten, continued avoidance of these foods seems a more likely explanation.</p> <p>Thoughtful readers may also wonder just how much insulin K was producing, at the time of her diagnosis, and just how many carbohydrates a thirty pound child needs to be healthy? It may be that GABA supplements and other chemical miracles will be unnecessary for large numbers of children who suffer from type 1 diabetes. Perhaps early diagnosis and permanent dietary adjustments will be what is needed to facilitate complete recovery for many, perhaps most, children afflicted by this insidious condition. Perhaps this case history will provide the necessary impetus to encourage undertaking controlled studies of dietary factors early in the disease process of type 1 diabetes.<br/>* While there are no carbohydrates that are essential to good health, there are essential <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;amino acids&lt;/b&gt;&lt;br&gt;&lt;br&gt;Organic acids containing the amino group NH2 which can react chemically as either an acid or a base.’); return false”>amino acids</a> and essential fats.</p> <p>Sources:</p> <ol><li>de la Monte SM, Wands JR. Alzheimer’s disease is type 3 diabetes-evidence reviewed. J Diabetes Sci Technol. 2008 Nov;2(6):1101-13.</li> <li><a href=”http://www.medicine.uottawa.ca/Students/MD/BlockOrientation/assets/documents/e_inf_week05.pdf” target=”_blank”>http://www.medicine.uottawa.ca/Students/MD/BlockOrientation/assets/documents/e_inf_week05.pdf</a></li> <li><a href=”http://www.elements4health.com/type-1-diabetes-patients-have-immune-response-to-wheat-proteins.html” target=”_blank”>http://www.elements4health.com/type-1-diabetes-patients-have-immune-response-to-wheat-proteins.html</a></li> <li>Scott FW, Sarwar G, Cloutier HE. Diabetogenicity of various protein sources in the diet of the diabetes-prone BB rat. Adv Exp Med Biol 1988; 246: 277–85.</li> <li>Scott F. Dietary initiators and modifiers of BB rat diabetes. In:Shafrir E, Renold AE, eds. Frontiers in Diabetes Research:Lessons from Animal Diabetes. London: Libbey, 1988: 34–9.</li> <li>Hoorfar J, Buschard K, Dagnaes-Hansen F. Prophylactic nutritional modification of the incidence of diabetes in autoimmune non-obese diabetic (NOD) mice. Br J Nutr 1993; 69: 597–607.</li> <li>Funda DP, Kaas A, Bock T, Tlaskalova-Hogenov H, Buschard K. Gluten-free diet prevents diabetes in NOD mice. Diabetes Metab Res Rev 1999; 15: 323–7.</li> <li>Bao F, Yu L, Babu S et al. One third of <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;HLA&lt;/b&gt;&lt;br&gt;&lt;br&gt;Human Leukocyte Antigen – elicit the strongest immunologic response in the body, and chromosome 6 is the genetic region that codes for these antigens. The genes that encode the class II molecules DQ2 and DQ8, the key genetic risk factors in celiac disease. Because the HLA complex is inherited intact as two haplotypes (one from each parent), siblings have 1 in 4 chance of being HLA-identical.’); return false”>HLA</a> DQ2 <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;homozygous&lt;/b&gt;&lt;br&gt;&lt;br&gt;Having identical genes at corresponding chromosomal loci, for example two fruit flies that are homozygous for red eye color.’); return false”>homozygous</a> patients with type 1 diabetes express celiac disease-associated transglutaminase autoantibodies. J Autoimmun 1999; 13:143–8.</li> <li>Lampasona V, Bonfanti R, Bazzigaluppi E et al. <a class=”HelpLink” href=”javascript:void(0)” onclick=”showHelpTip(event, ‘&lt;b&gt;Antibodies&lt;/b&gt;&lt;br&gt;&lt;br&gt;High molecular weight proteins which are produced by specialized B cells in the lymph nodes after stimulation by an antigen which act specifically against the antigen in an immune response. They typically consist of four sub-units that include two heavy and two light chains. Also known as immunoglobulin.’); return false”>Antibodies</a> to tissue transglutaminase C in type I diabetes. Diabetologia 1999; 42: 1195–8.</li> <li>Pocecco M, Ventura A. Coeliac disease and insulin-dependent diabetes mellitus: a causal association? Acta Paediatr 1995; 84: 1432–3.</li> <li>Hansen D, Brock-Jacobsen B, Lund E, Bjørn C, Hansen LP, Nielsen C, Fenger C, Lillevang ST, Husby S. Clinical Benefit of a Gluten-Free Diet in Type 1 Diabetic Children With Screening-Detected Celiac Disease A population-based screening study with 2 years’ follow-up Diabetes Care 29:2452-2456, 2006</li> <li>Soltani N, Qiu H, Aleksic M, Glinka Y, Zhao F, Liu R, Li Y, Zhang N, Chakrabarti R, Ng T, Jin T, Zhang H, Lu WY, Feng ZP, Prud’homme GJ, Wang Q. GABA exerts protective and regenerative effects on islet beta cells and reverses diabetes.Proc Natl Acad Sci U S A. 2011 Jul 12;108(28):11692-7. Epub 2011 Jun 27.</li> <li>Bouzane B, Postmedia News June 28, 2011</li> <li>Ford RP. The gluten syndrome: a neurological disease. Med Hypotheses. 2009 Sep;73(3):438-40. Epub 2009 Apr 29.</li> </ol><p>Celiac.com welcomes your comments below (registration is NOT required).</p> <br/><div class=”ArticleExtra”> <h2><strong>Related Articles</strong></h2> <ul><li><a href=”https://www.celiac.com/articles/24580/1/Parkinsons-Disease-Seizures-Slow-Recovery-from-Concussion/Page1.html” target=”_blank”>Parkinson’s Disease, Seizures, Slow Recovery from Concussion</a> <small>So far, 2014 has been a challenging new year for me…. <a href=”https://www.celiac.com/articles/24580/1/Parkinsons-Disease-Seizures-Slow-Recovery-from-Concussion/Page1.html#article”>[READ MORE]</a></small><br/></li> <li><a href=”https://www.celiac.com/articles/24494/1/Doctor-How-Do-I-Feel/Page1.html” target=”_blank”>Doctor, How Do I Feel?</a> <small>A surprising research report from Australia that explores non celiac gluten sensitivity (1) has given rise to a number of journalistic offerings that range between offensive and downright silly, while the reporters who wrote them appear to have somewhat compromised reading skills (3, 4)…. <a href=”https://www.celiac.com/articles/24494/1/Doctor-How-Do-I-Feel/Page1.html#article”>[READ MORE]</a></small><br/></li> <li><a href=”https://www.celiac.com/articles/24378/1/Is-a-Low-Glycemic-Paleo-Diet-Beneficial-for-Celiacs/Page1.html” target=”_blank”>Is a Low-Glycemic Paleo Diet Beneficial for Celiacs?</a> <small>It’s never become so clear to me how much our health and quality of life are dependent upon the food we eat since seeing myself, my family and more than my share of celiac friends and acquaintances make the transition to grain-free from gluten-free…. <a href=”https://www.celiac.com/articles/24378/1/Is-a-Low-Glycemic-Paleo-Diet-Beneficial-for-Celiacs/Page1.html#article”>[READ MORE]</a></small><br/></li> <li><a href=”https://www.celiac.com/articles/24136/1/Beware-the-Gluten-Free-Diet/Page1.html” target=”_blank”>Beware the Gluten-Free Diet</a> <small>Y Net News, under their “Health &amp; Science” banner, published an article titled “Israeli researchers propose link between gluten and ALS”, on April 17, 2015 (1)…. <a href=”https://www.celiac.com/articles/24136/1/Beware-the-Gluten-Free-Diet/Page1.html#article”>[READ MORE]</a></small><br/></li> </ul></div> <br/><p><strong><a href=”https://blockads.fivefilters.org”></a></strong> <a href=”https://blockads.fivefilters.org/acceptable.html”>(Why?)</a></p> Tue, 24 Jan 2017 19:30:00 +0000 no@spam.com (Dr. Ron Hoggan, Ed.D.) Type 1 Diabetes – A Case Study Supporting Integration of Existing Hypotheses – Celiac.com Article Diabetes is a condition in which blood glucose rises high enough to cause: damage to blood vessel walls, neurological injury, vision loss, and a host of other maladies. Most currently recognized cases of diabetes fall into one of two categories which are identified as type 1 and type 2 diabetes. https://www.celiac.com/articles/24660/1/Type-1-Diabetes–A-Case-Study-Supporting-Integration-of-Existing-Hypotheses/Page1.html https://www.celiac.com/content_images/diabetes_CC–Jill_Brown_thumb.jpg sv text/html https://www.celiac.com/articles/24660/1/Type-1-Diabetes–A-Case-Study-Supporting-Integration-of-Existing-Hypotheses/Page1.html https://www.celiac.com/articles/24655/1/Many-College-Students-Struggle-with-Gluten-free-Diet-on-Campus/Page1.html https://www.celiac.com/articles/24655/1/Many-College-Students-Struggle-with-Gluten-free-Diet-on-Campus/Page1.html <div readability=”39.330490405117″><span><a valign=”absmiddle” href=”https://www.celiac.com/articlerss/author/2″ target=”_blank”><img src=”https://www.celiac.com/templates/Gryphon/Images/icon_Rss.gif” border=”0″/></a> <span><a align=”left” valign=”absmiddle” href=”https://www.celiac.com/articlerss/author/2?podcastonly=1″ target=”_blank”><img src=”https://www.celiac.com/templates/Gryphon/Images/podcast.gif” border=”0″/></a></span></span> <h3>Jefferson Adams</h3> <img src=”https://www.celiac.com/authorpics/80c8b36ed4cc41e340843dab42f796f9.jpg” class=”Picture” border=”0″ align=”left”/><p>Jefferson Adams is a freelance writer living in San Francisco. His poems, essays and photographs have appeared in Antioch Review, Blue Mesa Review, CALIBAN, Hayden’s Ferry Review, Huffington Post, the Mississippi Review, and Slate among others.</p> <p>He is a member of both the National Writers Union, the International Federation of Journalists, and covers San Francisco Health News for <a href=”http://www.examiner.com/health-news-in-san-francisco/jefferson-adams” target=”_blank”>Examiner.com</a>.</p> <a href=”https://www.celiac.com/authors/2/Jefferson–Adams”>View all articles by Jefferson Adams</a></div><div itemprop=”articlebody” readability=”52.249336870027″> <div itemprop=”image”><span class=”FeatureImageSpanArticle”><img src=”https://www.celiac.com/content_images/smu–cc–daniel_lobo_thumb.jpg” property=”og:image” class=”Picture” width=”600″ height=”450″/></span><br/><span class=”FeatureImageSpanArticle”>Gluten-free diets can be hard to maintain on college campuses. Photo: CC–Daniel Lobo</span></div> <p>Celiac.com 01/24/2017 – Coming from homes where gluten-free food is abundant and taken for granted, many college students struggle with maintaining their diets during their time on campus.</p> <p>That struggle is the focus of numerous efforts by campuses nationwide to provide solid, reliable and abundant gluten-free food options for their students.</p> <p>At a place like SMU, that can include kitchen dining halls that serve gluten-free foods, or gluten-free pantry in Umphrey Lee.</p> <p>To help students be more conscious about their food choices SMU posts the daily menus on its website, along with nutritional facts for each item. There are different icons such as Eat Well, Fat Free, Low Sodium, Vegetarian, and Vegan, but as yet, no Gluten-Free icon.</p> <p>SMU does offer students access to a campus dietitian, who can help them figure out how to eat a balanced diet on campus, and grant them access to the gluten-free pantry or help in special cases.</p> <p>Read more at: <a href=”http://www.smudailycampus.com/ae/students-struggle-adhering-to-gluten-free-diet-on-campus” target=”_blank”>smudailycampus.com</a>.</p> <p>Celiac.com welcomes your comments below (registration is NOT required).</p> <br/><div class=”ArticleExtra”> <h2><strong>Related Articles</strong></h2> <ul><li><a href=”https://www.celiac.com/articles/24242/1/University-of-Delaware-Debuts-New-Gluten-friendly-Dining-Hall/Page1.html” target=”_blank”>University of Delaware Debuts New Gluten-friendly Dining Hall</a> <small>The drive to introduce specialties like kosher, gluten-free, vegan, and allergy-friendly foods at college campuses has really taken off in the last few years, with more and more colleges establishing alternative dining halls and food selections on their campuses…. <a href=”https://www.celiac.com/articles/24242/1/University-of-Delaware-Debuts-New-Gluten-friendly-Dining-Hall/Page1.html#article”>[READ MORE]</a></small><br/></li> <li><a href=”https://www.celiac.com/articles/24028/1/Elite-Colleges-Treat-Gluten-free-Students-to-Exclusive-Eateries/Page1.html” target=”_blank”>Elite Colleges Treat Gluten-free Students to Exclusive Eateries</a> <small>Gluten-free students at two elite liberal arts colleges in suburban Philadelphia, Pennsylvania, are now able to enjoy exclusive gluten-free dining areas…. <a href=”https://www.celiac.com/articles/24028/1/Elite-Colleges-Treat-Gluten-free-Students-to-Exclusive-Eateries/Page1.html#article”>[READ MORE]</a></small><br/></li> <li><a href=”https://www.celiac.com/articles/23602/1/The-Educational-Social-and-Family-Challenges-of-Children-with-Celiac-Disease-What-Parents-Should-Know/Page1.html” target=”_blank”>The Educational, Social, and Family Challenges of Children with Celiac Disease: What Parents Should Know</a> <small>This article originally appeared in the Summer 2013 issue of Journal of Gluten Sensitivity…. <a href=”https://www.celiac.com/articles/23602/1/The-Educational-Social-and-Family-Challenges-of-Children-with-Celiac-Disease-What-Parents-Should-Know/Page1.html#article”>[READ MORE]</a></small><br/></li> <li><a href=”https://www.celiac.com/articles/23512/1/Lehigh-University-Touts-New-Gluten-free-Dining-Options/Page1.html” target=”_blank”>Lehigh University Touts New Gluten-free Dining Options</a> <small>To create a gluten-free, allergen-free station in a dining hall that serves about 10,000 to 14,000 students each week, and offers a different daily menus for each meal, Lehigh University in Bethlehem went the distance…. <a href=”https://www.celiac.com/articles/23512/1/Lehigh-University-Touts-New-Gluten-free-Dining-Options/Page1.html#article”>[READ MORE]</a></small><br/></li> </ul></div> <br/></div><p><strong><a href=”https://blockads.fivefilters.org”></a></strong> <a href=”https://blockads.fivefilters.org/acceptable.html”>(Why?)</a></p> Tue, 24 Jan 2017 16:30:00 +0000 no@spam.com (Jefferson Adams) Many College Students Struggle with Gluten-free Diet on Campus – Celiac.com Article Coming from homes where gluten-free food is abundant and taken for granted, many college students struggle with maintaining their diets during their time on campus. https://www.celiac.com/articles/24655/1/Many-College-Students-Struggle-with-Gluten-free-Diet-on-Campus/Page1.html https://www.celiac.com/content_images/smu–cc–daniel_lobo_thumb.jpg cy text/html https://www.celiac.com/articles/24655/1/Many-College-Students-Struggle-with-Gluten-free-Diet-on-Campus/Page1.html

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